By Dr. L. F. Agnati, O. Franzen, S. Ferré, G. Leo (auth.), Dr. R. Horowski, Prof. Y. Mizuno, Prof. C. W. Olanow, Prof. W. H. Poewe, Prof. P. Riederer, Dr. J. A. Stoessl, Prof. M. B. H. Youdim (eds.)
• attainable position of intramembrane receptor-receptor interactions in reminiscence and studying through formation of long-lived heteromeric complexes: specialise in motor studying within the basal ganglia (L.F. Agnati, O. Franzen, S. Ferré, G. Leo, R. Franco, ok. Fuxe) •Self-tolerance within the immune privileged CNS: classes from the entorhinal cortex lesion version (E. Kwidzinski, L. ok. Mutlu, A. D. Kovac, J. Bunse, J. Goldmann, J. Mahlo, O. Aktas, F. Zipp, T. Kamradt, R. Nitsch, I. Bechmann) •Progressive neurodegeneration in Drosophila: a version process (J.-A. Tschäpe, A. Bettencourt da Cruz, D. Kretzschmar) •Effect of complex glycation endproducts on mobilephone cycle and their relevance for Alzheimer`s disorder (G. Münch, J. Gasic-Milenkovic, T. Arendt) •What have we learnt from cDNA microarray gene expression reports concerning the position of iron in MPTP triggered neurodegeneration and Parkinson`s disorder? (M. B. H. Youdim) •Animal types of Parkinson`s disorder in rodents brought on through pollutants: an replace (E. C. Hirsch, G. Höglinger, E. Rousselet, T. Breidert, okay. Parain, J. Feger, M. Ruberg, A. Prigent, C. Cohen-Salmon, J.-M. Launay) •General facets of neurodegeneration (K. A. Jellinger) •The neuromelanin of human substantia nigra: constitution, synthesis and molecular behaviour (L. Zecca, F. A. Zucca, P. Costi, D. Tampellini, A. Gatti, M. Gerlach, P. Riederer, R. G. Fariello, S. Ito, M. Gallorini, D. Sulzer) •New strategies and instruments in imaging for the learn of neurodegenerative illness (A. J. Stoessl, C. S. Lee, R. de l. a. Fuente-Fernandez) •Arguments for using dopamine receptor agonists in medical and preclinical Parkinson`s ailment (M. Gerlach, ok. Double, H. Reichmann, P. Riederer) •The function of acetylcholine and dopamine in dementia and psychosis in Parkinson`s sickness (J. L. W. Bosboom, D. Stoffers, E. Ch. Wolters) •Growth/differentiation factor-15 (GDF-15), a singular member of the TGF-ß superfamily, promotes survival of lesioned mesencephalic dopaminergic neurons in vitro and in vivo and is prompted in neurons following cortical lesioning (J. Strelau, A. Schober, A. Sullivan, L. Schilling, okay. Unsicker) •Gene treatment for Parkinson`s sickness (H. Mochizuki, Y. Mizuno) •Stereotaxic intrastriatal implantation of human retinal pigment epithelial (hRPE) cells hooked up to gelatin microcarriers: a possible new phone remedy for Parkinson`s sickness (R. L. Watts, C. D. Raiser, N. P. Stover, M. L. Cornfeldt, A. W. Schweikert, R. C. Allen, T. Subramaniam, D. Doudet, C. R. Honey, R. A. E. Bakay)
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Extra info for Advances in Research on Neurodegeneration: Volume 10
Indications for heteromerization. Neuropsychopharmacology 23: 50-59 Franzen 0, Lennerstrand G, Pardo J, Richter H (2000) Spatial contrast sensitivity and visual accommodation studied with VEP (Visual Evoked Potential), PET (Positron Emission Tomography) and psychophysical techniques. In: Franzen 0, Richter H, Stark J (eds) Accommodation and convergence mechanisms in the visual system. Birkhauser, Basel, pp 91-114 Frauenfelder H, Sligar SG, Wolynes PG (1991) The energy landscapes and motions of proteins.
Nitschl, and I. Bechmann1 2 Departments of 1 Cell and Neurobiology and Institute of Neuroimmunology, Institute of Anatomy, Charite, Medical Faculty, Humboldt-University, and 3 Deutsches Rheumaforschungszentrum, Berlin, Germany Summary. Upon peripheral immunization with myelin epitopes, susceptible rats and mice develop T cell-mediated demyelination similar to that observed in the human autoimmune disease multiple sclerosis (MS). In the same animals, brain injury does not induce autoimmune encephalomyelitis despite massive release of myelin antigens and early expansion of myelin specific T cells in local lymph nodes, indicating that the self-specific T cell clones are kept under control.
The suggested view, while indirectly supported by results obtained also by other researchers, is intended to be mainly a heuristic hypothesis of some use to further proceed in the understanding of the functional interplay between basal ganglia and cerebral cortex in the control of the two main aspects of motor behaviour: the control of the execution of movements and the formation and storage of new motor programs. We are fully aware that the challenge in learning and memory (and more generally in the understanding of the integrative action of the brain) is to achieve in-putting in one and the same coherent picture intrinsically different data, and to develop a logical frame that allows a transit from molecular neurobiology, via neurophysiology, to neuropsychology.